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Los avances más significativos en la fisiopatología de la fibrosis hepática que se presentaron en la DDW 2006 fueron: S1595. Imbalance between angiotensin II type 1 and 2 receptor signals is a key factor in pathogenesis of liver fibrosis: A novel target for therapeutic strategy of fibrotic liver diseases in metabolic syndome. S1596. Keratin mutation predisposes to thioacetamide- but not CCl4-induced mouse liver fibrosis S1597. Gc-globulin concentrations modify the profibrogenic effect of at-risk haplotypes on serum activities of complement factor C5 142. Hepatic stellate cell production of resistin modulates liver insulin sensitivity 144. S-adenosylhomocysyeine (SAH) may sensitize to TNF-induced liver injury in alcoholic liver disease patients with impaired mitochondrial methionine metabolism PARA ESTA INFORMACIÓN, SUGERIMOS DESCARGAR FORMATO PDF.

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2006-11-08   |   1,526 visitas   |   Evalua este artículo 0 valoraciones

Vol. 71 Núm.1. Agosto 2006 Pags. 120-121 Rev Gastroenterol Mex 2006; 71(Supl. 1)